Vasopressors (artery and venous), Catecholamines (epinephrine, norepinephrine, dopamine, phenylephrine) these Stimulate α1 (↑ SVR/vasoconstriction) and/or β1 (↑ contractility/HR) receptors VS Non-catecholamines (vasopressin, angiotensin II): Cause vasoconstriction via non-adrenergic pathways.
spectrum= pure alpha 1--> mixed--> pure beta 1
Phenylephrine (Pure α1)
AE: Reflex bradycardia , not with AR
0.25-3 mcg/kg/min (titreate by 0.25)
PIV Maximum dose: 0.5 mcg/kg/min
NoreEpi/Levophed (α1>>>β1)
AE: Peripheral ischemia (digits, gut), Extravasation → tissue necrosis
α1 → vasoconstriction → ↑ SVR and β1 → mild ↑ contractility
0.05 - 1 mcg/kg/min (titrate by 0.03)
PIV Maximum dose: 0.2 mcg/kg/min
put central line if ≥0.1–0.2, PIV 18-20 gage ok, Maximum dose (PIV): 0.2 mcg/kg/min
also good place to add 2nd agent when 0.15-0.2
Epi (β1 (LD)> α1 (HD), also β2 )
AE: Tachyarrhythmias, ↑ lactate (β2 effect, not always hypoperfusion), Hyperglycemia, arrhythmia
0.05-0.5 mcg/kg/min (titrate by o.02) (( 0.05-0.2 has more isotropy, up to 0.3 more vasoconstriction ))
Maximum dose (PIV): 0.1 mcg/kg/min
Watch: HR + lactate trend (can be misleading)
Dopamine (Mod-β1, High-α1 ) (X)
AE: Tachyarrhythmias (big one) arrhythmia
2-20 mcg/kg/min (titrate by 2)
Medium (3–10 mcg/kg/min)→ β1 (contractility), High (>10 mcg/kg/min) → α1 (vasoconstriction)
Start 5 mcg/kg/min Titrate by 2–5 mcg/kg/min every few minutes , Usual range: 5–20 mcg/kg/min
PIV Max dose 10 mcg/kg/min
Watch: AFib, VT
-Vasopressin (add-on)
AE: Ischemia (skin, gut) Hyponatremia (V2 effect at higher doses)
V1 (vascular) Vasoconstriction
fixed dose 0.03 or 0.04, Always in CVC never PIV, Never alone it is add on
Methylene blue or hydroxocobalamin (vasodilator scavenger)
when 3rd vasopressor is added
methaleen blue turn urine bright green color ,
avoid in G6PD and on other seretonin syndrom increasing drugs
hydroxocobalamin another option makes urine dark red ( hard for hemodylaissi)
INOTROP
-Dobutamine (β1 → contractility >> β2 →vasodilation)
(Easier to titrate, less Hypotn)
AE: arrhythmias ,Hypotension (β2 vasodilation)
β1 → ↑ contractility and β2 → mild vasodilation, start NEPI first then add this
5-40 mcg/kg/hr
Watch: BP drop while HR rises
Milrinone (Pulmonary HTN / RV failure, more HypoTn, renal Clarence) same add NEPI first
Methylene blue:
Causes blue-green urine discoloration
Avoid in G6PD deficiency
Use caution with serotonergic drugs (risk of serotonin syndrome)
Hydroxocobalamin:
Causes dark red urine discoloration
Can interfere with hemodialysis machines and lab assays
Wean norepinephrine first, then discontinue vasopressin; consider midodrine 5–10 mg TID as an oral bridge (delayed onset over hours), and discontinue prior to discharge.
Shock → Vasopressor Approach
Undifferentiated / Septic shock →
Norepinephrine ± vasopressin ± epinephrineCardiogenic shock →
Norepinephrine ± dobutamine (± milrinone) ± epinephrine ± vasopressin (adjunct)
→ If low CI → add epinephrine/dobutamine
→ If low BP → add vasopressinObstructive shock →
Treat cause + norepinephrine ± vasopressin ± epinephrineAnaphylactic shock →
Epinephrine (β2 + α1 effects)Post-epidural hypotension →
PhenylephrineLV outflow tract obstruction →
Phenylephrine ± vasopressin (avoid inotropes)
Perfusion Assessment
Lactate = only one data point
Always assess:
Urine output
Mentation
Liver function
Renal function
Skin perfusion
👉 Lactate = “check engine light,” not the whole picture
⚠️ Lactate may be falsely elevated with:
Albuterol / continuous nebs
Epinephrine
🧠 Push-Dose Pressors (Peri-intubation / crash)
Use when acute hypotension (e.g., intubation) BP<50
Intubation ↓ sympathetic tone + preload
Options:
Epinephrine: 20–50 mcg (esp bradycardia / low CI)
Phenylephrine: 100–200 mcg
± Vasopressin (less common push)
👉 Always:
Have pressors + 500 mL LR ready
Start infusion ASAP (push is temporary)
🧠 General Pressor Safety
Vasopressin → requires central line (preferred)
Catecholamines at low dose can be given peripherally (with monitoring + antidote available)
Before intubation:
Prepare push-dose pressors (premixed syringes)
Cycle BP frequently (q1 min)
Consider A-line
🧠 Pearls
Dopamine in crash carts → stable at room temp (logistical reason, not clinical superiority)