Severe Symptomatic Anemia
Anemia causing symptomatic impairment — iron deficiency, anemia of chronic disease, or CKD-related; requiring workup and often transfusion
Symptoms / Associated Sx
Fatigue, weakness, exertional dyspnea (most common)
Pallor (conjunctival, palmar, nail beds), tachycardia
Palpitations, chest pain/angina (demand ischemia)
Headache, difficulty concentrating
Pica (ice chewing = pagophagia — classic for iron deficiency), restless legs syndrome
Koilonychia (spoon nails), angular cheilitis, glossitis (iron deficiency — severe/chronic)
Denies
Acute hemorrhage event (rules out acute blood loss as primary etiology)
Fever, bone pain, lymphadenopathy (rules out hematologic malignancy if absent)
Jaundice, dark urine, back pain (rules out hemolytic anemia)
Family history of hemolytic or hereditary anemia disorders
Social History (SHx)
Dietary history (vegetarian/vegan → iron, B12 deficiency; alcohol → folate/B12), menstrual history (heavy periods → iron loss), chronic disease (CKD, IBD, malignancy, CHF, rheumatoid arthritis), prior GI workup, prior anemia diagnosis, renal replacement therapy.
Main Etiology
Iron deficiency anemia (IDA): Blood loss (GI, menstrual), inadequate intake, malabsorption (celiac, post-gastric surgery)
Anemia of chronic disease (ACD): Chronic inflammation (RA, IBD, malignancy, CKD) → hepcidin elevation → iron sequestration
CKD-related anemia: Decreased erythropoietin production + iron deficiency + uremic toxins
B12/folate deficiency: Dietary, malabsorption, pernicious anemia, methotrexate
Thalassemia trait: Microcytic anemia with normal/elevated ferritin; common in Mediterranean, Southeast Asian, African populations
Most Common DDx
Iron deficiency anemia (microcytic; low ferritin <30; low iron; high TIBC; low transferrin saturation; responds to iron — distinguish from ACD by ferritin level)
Anemia of chronic disease (normocytic or mildly microcytic; ferritin normal-to-high; low TIBC; underlying chronic illness; iron infusion less effective without treating underlying disease)
B12 deficiency (macrocytic MCV; hypersegmented neutrophils on smear; low serum B12; elevated MMA and homocysteine; neurologic symptoms — paresthesias, ataxia)
Folate deficiency (macrocytic; low serum folate; elevated homocysteine but normal MMA — distinguishes from B12; dietary or methotrexate-related)
Thalassemia trait (microcytic + low MCV but RBC count HIGH; normal ferritin; Mentzer index <13 suggests thalassemia; Hgb electrophoresis confirms)
Hemolytic anemia (elevated LDH, elevated indirect bilirubin, low haptoglobin; reticulocytosis; peripheral smear abnormal)
Aplastic anemia (pancytopenia; hypocellular bone marrow; no chronic disease needed)
DATA
CBC with differential (MCV — microcytic: IDA, thalassemia; normocytic: ACD, CKD; macrocytic: B12/folate)
Reticulocyte count + reticulocyte production index (RPI <2 = hypoproliferative; >2 = blood loss or hemolysis)
Iron studies: serum iron, TIBC, transferrin saturation, ferritin
B12, folate (if macrocytic or dietary risk)
Peripheral blood smear (morphology — hypochromic/microcytic: IDA; hypersegmented PMNs: B12/folate; schistocytes: hemolysis/TMA)
CMP (creatinine — CKD; LFTs; albumin)
Hemoglobin electrophoresis (if thalassemia suspected)
Reticulocyte Hgb (Chr) — most sensitive early marker of functional iron deficiency
CRP/ESR (ferritin is an acute phase reactant — can be elevated even with IDA in chronic inflammation)
EPO level (low in CKD anemia; elevated in other causes)
Stool FOBT / FIT test; GI workup (colonoscopy/EGD) if IDA in men or postmenopausal women
Home Meds
PPIs (reduce iron absorption — separate from iron supplements by 2h)
Metformin (B12 malabsorption with long-term use)
Methotrexate (folate antagonist — ensure folate supplementation)
ESA (erythropoiesis-stimulating agents — if on for CKD; assess compliance and dose)
NSAIDs (chronic GI blood loss)
Plan
Transfusion (if symptomatic and severe):
Threshold Hgb <7 (stable); <8 (cardiovascular disease, ACS, elderly symptomatic)
1 unit pRBCs at a time; reassess after each unit; do not transfuse asymptomatic stable patients to a "number"
Iron deficiency anemia:
Oral iron: ferrous sulfate 325 mg PO every other day (superior absorption vs. daily per recent evidence) — take on empty stomach; vitamin C enhances absorption; separate from PPIs by 2h
IV iron (preferred if: intolerant of PO, malabsorption, IBD, CKD, heart failure, ongoing blood loss, Hgb <8 with symptoms): Ferric carboxymaltose (Injectafer) 750 mg IV × 2 doses 7 days apart OR Iron sucrose 200 mg IV × 5 doses OR Low-molecular-weight iron dextran 1000 mg IV × 1 (total dose infusion)
Identify and treat underlying cause of iron deficiency (GI workup mandatory in men and postmenopausal women)
Anemia of chronic disease:
Treat underlying inflammatory condition (primary intervention)
IV iron if functional iron deficiency (transferrin saturation <20% despite normal/elevated ferritin)
ESA (erythropoiesis-stimulating agents — epoetin alfa or darbepoetin) only in CKD or cancer-related anemia; avoid in active malignancy outside of chemotherapy-related indications (increased mortality/thrombosis risk)
CKD-related anemia:
Iron repletion first: target ferritin >200–500 ng/mL and transferrin saturation >20% (IV iron preferred)
ESA therapy: Darbepoetin alfa 0.45 mcg/kg SQ/IV q4 weeks (or epoetin alfa 50–100 units/kg SQ/IV 3× weekly); target Hgb 10–11.5 g/dL (do NOT target Hgb >13 — increased cardiovascular events)
Nephrology co-management for ESA dosing and monitoring
B12 deficiency:
Cyanocobalamin 1000 mcg IM daily × 7 days → weekly × 4 weeks → monthly (pernicious anemia or malabsorption)
High-dose oral B12 2000 mcg PO daily (effective even without intrinsic factor via passive absorption)
Folate deficiency: Folic acid 1–5 mg PO daily × 1–4 months; always replace B12 first (or simultaneously) — folate without B12 can mask neurologic damage
Identify source of iron loss: colonoscopy + EGD in all men and postmenopausal women with IDA
Trend CBC, reticulocyte count, iron studies weekly (oral iron) or 2–4 weeks (IV iron)
PT/OT if deconditioning; dietitian consult if nutritional deficiency
Discharge: Iron supplementation (oral or IV based on severity/tolerance); B12/folate as appropriate; GI follow-up for source identification; nephrology if CKD-related; Hgb recheck in 2–4 weeks; treat underlying chronic disease
Red Flags
Hgb <7 + angina/chest pain → demand ischemia / Type 2 MI → EKG, troponin, transfuse to Hgb ≥8; cardiology
New-onset severe anemia in men or postmenopausal women → malignancy workup (colon cancer, gastric cancer) before attributing to benign cause
Pancytopenia + severe anemia → aplastic anemia or hematologic malignancy → bone marrow biopsy; hematology urgently
Neurologic symptoms (paresthesias, ataxia, cognitive decline) + macrocytic anemia → B12 deficiency → treat immediately (neurologic damage is irreversible if prolonged)
ESA in cancer patients outside of chemotherapy-related anemia → increased mortality and thrombosis — avoid
Senior IM Resident Pearls
Ferritin is an acute phase reactant — can be falsely normal or elevated in iron deficiency with coexisting inflammation (ACD + IDA); CRP >5 invalidates ferritin as a standalone iron marker; use transferrin saturation <20% as additional criterion
Mentzer index: MCV ÷ RBC count; <13 suggests thalassemia; >13 suggests IDA — useful screening tool when distinguishing microcytic anemia etiologies
Every-other-day oral iron is superior to daily dosing — reduces hepcidin surge that blocks absorption with consecutive daily doses (Moretti et al.)
ESA target Hgb 10–11.5 g/dL in CKD — targeting >13 increases cardiovascular events and mortality (TREAT, CHOIR trials); never normalize Hgb with ESA
Common mistake: Replacing folate without checking B12 — folate corrects macrocytic anemia but does not treat subacute combined degeneration of the spinal cord; neurologic damage progresses silently
Common mistake: Attributing iron deficiency in a 55-year-old male to "dietary" without colonoscopy — colorectal cancer must be excluded in all men and postmenopausal women with IDA
ORDERS
Labs
Admission
CBC with diff
CMP
BMP
Reticulocyte count + RPI
Iron studies (Fe, Ferritin, TIBC, TSAT)
Peripheral smear
Type & Screen
Type & Cross
Additional
Vitamin B12
Folate
CRP
ESR
LDH
Haptoglobin
Indirect bilirubin
Hemoglobin electrophoresis
EPO level (CKD)
Reticulocyte Hgb (Chr)
FOBT/FIT
Trending
CBC daily
Retic count q3–7 days
Iron studies q2–4 weeks after treatment
Hgb after each transfusion
Imaging
Iron Deficiency Workup
Colonoscopy
EGD
If Malignancy Concern
CT Chest
CT Abdomen/Pelvis
Procedures
Blood Transfusion
1 unit PRBC at a time
Recheck Hgb after each unit
Bone Marrow Biopsy
If:
Pancytopenia
Aplastic anemia concern
Hematologic malignancy concern
Medications
Transfusion
Hgb <7
Transfuse PRBC
Hgb <8
CAD
ACS
Elderly symptomatic
Active angina
Goal:
Symptom improvement
Not a specific number
Iron Deficiency
Oral Iron
Ferrous sulfate 325 mg PO every other day
Vitamin C 500 mg PO daily
IV Iron (Preferred if severe)
Ferric Carboxymaltose
750 mg IV x2 doses (7 days apart)
OR
Iron Sucrose
200 mg IV x5 doses
OR
Iron Dextran
1000 mg IV x1
B12 Deficiency
Cyanocobalamin
1000 mcg IM daily x7 days
Then weekly x4 weeks
Then monthly
OR
B12 2000 mcg PO daily
Folate Deficiency
Folic Acid
1–5 mg PO daily
Always replace B12 first or simultaneously.
CKD Anemia
Darbepoetin Alfa
0.45 mcg/kg SQ q4 weeks
OR
Epoetin Alfa
50–100 units/kg SQ/IV TIW
Target:
Hgb 10–11.5
Never target >13.
Hold
NSAIDs
Excessive phlebotomy
PPIs if possible (reduce iron absorption)
Methotrexate without folate replacement
Consults
Gastroenterology
All men with IDA
Postmenopausal women with IDA
FOBT positive
Hematology
Severe anemia
Pancytopenia
Hemolysis
Unclear etiology
ESA management
Nephrology
CKD anemia
ESA initiation
Cardiology
If:
Chest pain
Elevated troponin
Demand ischemia
Nutrition
Nutritional deficiencies
PT/OT
Weakness
Deconditioning
Nursing
Telemetry if severe anemia
Fall precautions
Strict I&O
Orthostatic vitals
Monitor transfusion reactions
Notify physician:
Chest pain
Syncope
New hypoxia
Hgb drop >1 g/dL
Active bleeding
Follow-Up
CBC daily
Iron studies after treatment
Retic response in 1 week
Colonoscopy results
EGD results
Hemolysis labs
B12/Folate results
Expected response:
Retic rise: 3–5 days
Hgb rise: ~1 g/dL per unit PRBC
Iron therapy: Hgb rise ~1–2 g/dL every 2–3 weeks