POOR ORAL INTAKE / DEHYDRATION
Inadequate fluid/caloric intake leading to intravascular volume depletion — common precipitant of AKI, electrolyte disorders, delirium, and falls
SYMPTOMS / ASSOCIATED SX
Thirst, dry mouth, dark/concentrated urine, decreased urine output (<0.5 mL/kg/h)
Orthostatic lightheadedness, presyncope, syncope
Fatigue, lethargy, AMS (severe)
Exam: dry mucous membranes, tachycardia, orthostatic hypotension, decreased skin turgor (unreliable in elderly)
DENIES
Chest pain/dyspnea (cardiogenic — not dehydration)
Fever/diarrhea/vomiting (identify GI/infectious precipitant)
Dysphagia (mechanical cause — NPO without evaluation)
SOCIAL HISTORY
Ability to access and prepare fluids independently; cognitive status; caregiving situation
Alcohol use; recent vomiting or diarrheal illness
MAIN ETIOLOGY
Reduced intake: AMS, dysphagia, reduced thirst sensation (elderly), NPO/fasting, functional dependence
GI losses: vomiting, diarrhea, NGT drainage, high-output ostomy/fistula
Insensible losses: fever, burns, sweating, hyperventilation
Renal losses: DI (central or nephrogenic), osmotic diuresis (DKA), diuretics
Third-spacing: ascites, pleural effusion, bowel obstruction, burns
MOST COMMON DDX
AKI prerenal (BUN:Cr >20; FeNa <1%; responds to fluids)
Hypernatremia (Na >145 — free water deficit; coexists with dehydration in elderly)
DKA/HHS (hyperglycemia, ketonemia, osmotic diuresis)
Central/nephrogenic DI (polyuria, urine specific gravity <1.005)
Adrenal insufficiency (hypoNa + hyperK + hypotension)
Sepsis (third-spacing, reduced intake)
DATA
BMP (BUN:Cr >20 = prerenal); urine Na + Cr → FeNa = (urine Na × plasma Cr)/(plasma Na × urine Cr) × 100
Urine specific gravity; serum osmolality; orthostatic VS
Weight trend (1 kg ≈ 1 L fluid); strict I&Os; urine output
CBC (hemoconcentration); glucose; HbA1c; bladder scan if oliguria unexplained
HOME MEDS
Diuretics — hold until euvolemic; reassess dose
ACE inhibitors/ARBs — hold if AKI or hypotension
NSAIDs — hold (worsen AKI)
Metformin — hold if AKI or contrast planned; digoxin — hold (levels rise with dehydration)
PLAN
Fluid selection (SMART and SALT-ED trials — prefer balanced crystalloids for large-volume resuscitation):
Isotonic depletion: LR or PlasmaLyte preferred over NS (lower hyperchloremic acidosis risk)
Hypovolemia + hyponatremia: NS 0.9% initially; correct Na no faster than 6–8 mEq/L/24h
Hypovolemia + hypernatremia: D5W or 0.45% NaCl; correct no faster than 10–12 mEq/L/24h
Severe hemodynamic instability: NS FIRST to restore perfusion, then switch to hypotonic/balanced
IV bolus: 500 mL–1 L LR or NS; reassess after each bolus; maintenance 125–150 mL/h after correction
Electrolyte replacement as needed (K, Mg, Phos)
Oral route preferred when tolerated; speech therapy if dysphagia; ORT for mild-moderate dehydration
Antiemetics: ondansetron 4 mg IV q6h PRN; loperamide 4 mg then 2 mg after loose stool (non-infectious only)
Target urine output 0.5–1 mL/kg/h
DISCHARGE:
Ensure oral intake adequate (≥1.5–2 L/day) before discharge
Restart held meds only after renal function confirms safety
BMP in 3–5 days if electrolyte abnormalities at discharge
RED FLAGS
SBP <90 + tachycardia + oliguria → hypovolemic shock; 1–2 L IV bolus rapidly; ICU if no response
Anuria + no response to fluid bolus → obstruction or intrinsic AKI; bladder scan; nephrology
Dysphagia + aspiration → NPO; speech therapy urgently
Large-volume NS (>3 L): monitor for hyperchloremic metabolic acidosis; switch to balanced crystalloid
SENIOR IM RESIDENT PEARLS
SMART trial (NEJM 2018): balanced crystalloids reduced MAKE vs. NS in ICU patients
SALT-ED trial (NEJM 2018): similar findings in non-ICU — shift to balanced crystalloids in standard practice
BUN:Cr >20 = prerenal but poor sensitivity/specificity; FeNa <1% more specific but unreliable on diuretics — use FeUrea <35% instead
Common mistake: giving NS to all dehydrated patients regardless of Na — check Na before choosing fluid
ORT (WHO solution): sodium 75 mEq/L + glucose 75 mmol/L — equivalent to IV in mild-moderate dehydration