Hypoxemia-Respiratory Distress
Initial stuff
sick/not sick → work of breathing? (accessory muscles, tripoding) will tire it will get worse, are they protecting their airway, responsive (unless 2 <80 wil max NPPV) vs unresponsive (Lethargic, obtunded, vomiting.) ? → if unresponsive need intubation → icu? (code status)
position/sit pt up/apply Oxygenation → then DDX
how Hypoxemia (saturation)? if too low jump in to bipap, Sign of problems with oxygen delivery? (AMS, lactate, chest pain, cyanosis)?
vitals and I/O and last labs?
look at chart Hx (HF,stroke,delerium,COPD,PNA) main issue of admission, talk to RN about event, recent interventions, medalist (resp depresion, AC), transfusion or fluid? last CXR/CT/Echo
physical exam (HF, wheezing, crackles, DVT) — not most useful → POCUS (PTX,Aline-N/Bline-fluid,effusion,systolic fxn,DVT)
DDX
Alveolar
Pulm edema (HF exac, falsh pulm edema (tachyarrythmia, ACS, HTN-E,TACO,TRALI)) (crackles,JVD,Edema,on IVFs/blood,)
Aspiration Pneumonitis (fast) /PNA (hr-days) (chocking,Vomiting evidence)
Effusion
Airway
COPD/Astma exac
anaphylaxis
angioedema
mucus plugging.
Other
absent lung sound → PTX (truma), Atelectasis, large effusion (Hf,Cirrhosis, empeima (fever,WBC))
PE (no CXR finding, Normal exam, tachy)
DX
ABG (pulse Ox not always accurate) PaO2/PCo2, trend
CC panel (lactate,)
CBC
BMP
trop
CXR
EKG
echo if HF exac pr Flash
TX
call RT
sit upright, suction
O2 > 90 → NC → non-rebreather → HFNC → NIPP → intubate
pulmonary edema (volume overload, HF) → stop IVF/blood → 80 IV Lasix → CPAP (normo-/hypocapnic PaCO₂ ≤45) or BiPAP hypercapnic (PaCO₂ >45, acidosis)
flash pulmonary edema → nitro
aspiration pneumonitis/PNA → HFNC (caution CPAP/BiPAP), start Abx if (≥2/3: fever/WBC/purulent sputum + new infiltrate)
cardiogenic pulmonary edema – High-dose NTG: If SBP <160 → 800 mcg IV over 2–3 min, repeat q3–5 min (total 2–3 mg). If SBP ≥160 → 1–2 mg IV over 3–5 min, repeat q3–5 min ×2–3 doses. Then start infusion 100–400 mcg/min targeting SBP ↓20–30%.
wheezing → Duoneb nebulizer
absent lung sounds → PTX, atelectasis, large effusion (C-tube if large PTX and only NC-NRB; HFNC and NIPPV are contraindicated). For atelectasis (x-ray one side white out and trachea shift toward → CPT, frequent suctioning, breathing treatment, pulm). Effusion (x-ray one side white out and trachea shift away → think effusion [Lasix], empyema [tube + Abx], PTX [tube])
PE (CTA, US, AC)
NPO until stable
Note:
[[hypoxemia]] low O2 in blood (pulse ox, blood gas)
hypoxia: low O2 at the level of cells and tissue
oxygen content: saturation + Hgb level
oxygen delivery: oxygen content × CO → how much is pumped to tissue
pulse ox: measures what saturation of Hgb is
blood gas: measures amount of dissolved O2 → PaO2
not protecting airway: not responsive to stimuli, can see vomit or nosebleed and they are gurgling, frequent suctioning
pulse ox is a time machine; it will take 30 seconds to change
in V/Q mismatch, when part of lung is not getting as much ventilation, a lot of O2 should correct it; when part of the lung is getting no ventilation like in shunt, then O2 won’t fix it
A-a difference from blood gas (alveolar O2 − arterial O2) – MedCalc – normal (5–10, 20 in older) means low inspired O2 or alveolar hypoventilation; rest of issues have higher A-a gradient (3,4,5,6); don’t need it for everyone unless can’t find why
NC – 1–6 L/min, 3% additional FiO2 per L, up to like 45% (O2 100% plus environment 21%)
venti mask (no bag on it) – blowing 10 L/min around FiO2 like 45–50% (O2 100% plus environment 21%)
non-rebreather mask, has bag on it, bag inflated, less from environment, FiO2?
HFNC (heated), can choose FiO2 and flow rate 10–60 L/min, up to 100% FiO2; if can’t tolerate NIPP mask or high risk for aspiration; use max flow rate and FiO2 in rapid situations
NIPP – (different interfaces), continuous (CPAP) or non-continuous (BiPAP) positive pressure; CPAP keeps airway open and improves oxygenation like in OSA, keeps trachea open; BiPAP in HF with fluid in alveoli (popping when opening/closing), positive pressure keeps alveoli open and improves gas exchange and helps blow out CO2 (like if work of breathing is high); two pressures are set: EPAP and IPAP triggered by each breath (can max FiO2 to 100%)
not protecting airway is absolute contraindication for NIPP (if vomit will aspirate); they need to be awake and not in restraints for NIPP, or RT needs to be there with them
BiPAP → if hypercapnia, COPD, OHS; 10 cm H2O good start for EPAP and IPAP 5 cm H2O above EPAP, around 15
CPAP → if hypoxia without hypercapnia, like in respiratory distress and increased work of breathing; 10 cm H2O good CPAP value
volume overload → HF or NIPP; if awake NIPP (HF, COPD while meds work)
position is important; sit the patient up
ABG analyzing → if blood doesn’t come out quickly might be mixed; put the right cap on to take the air out; if frothy that is worthless; look at all the numbers including methemoglobin and carboxyhemoglobin
Framework (not realistic in RRT)
Low inspired O2 (on mountain) – normal A-a
Alveolar hypoventilation: both hypoxemia and hypercarbia, central (opioid, OHS, brainstem stroke, paralysis) or peripheral (airflow obstruction) – normal A-a
V/Q mismatch: (atelectasis, ) better with O2
Shunt – not better with O2
Diffusion limitation (rare at rest) → alveolar-capillary thickness. When CO is elevated and blood is moving fast, RBCs don’t have time to get oxygen; usually fibrosis and edema; they desaturate with movement, not at rest
Low mixed venous O2 (tissue taking most O2, can’t re-oxygenate fully), like severe anemia, low CO – body takes most of the oxygen
If you'd like, I can also make this into a clean printable ICU one-page reference sheet.