Venous Thromboembolism (VTE) — DVT / PE
Symptoms / Associated Sx
DVT: Unilateral leg swelling, erythema, warmth, tenderness (calf or thigh); Homan's sign (unreliable)
PE: Dyspnea (most common), pleuritic chest pain, tachycardia, hypoxia, cough, hemoptysis
Massive PE: Syncope, hypotension, cardiac arrest (obstructive shock)
Low-grade fever; elevated JVP, right heart strain signs (massive/submassive PE)
Denies
Bilateral leg swelling (rules out DVT as primary — raises heart failure, hypoalbuminemia, bilateral compression)
Fever + purulent wound (rules out cellulitis as primary cause of leg erythema/swelling)
Pleuritic pain with productive cough + fever + infiltrate (rules out pneumonia as primary)
Prior similar episode with negative workup (reduces VTE likelihood)
Social History (SHx)
Recent surgery (especially orthopedic, pelvic, abdominal), immobility, malignancy, prior VTE, oral contraceptives/HRT, pregnancy/postpartum, long-distance travel, hereditary thrombophilia (Factor V Leiden, prothrombin mutation, protein C/S deficiency, antithrombin deficiency), obesity, smoking.
Main Etiology
Virchow's triad: stasis (immobility, heart failure) + hypercoagulability (malignancy, thrombophilia, OCP/HRT, pregnancy) + endothelial injury (surgery, trauma, IV catheter)
Provoked VTE: surgery, trauma, immobility, malignancy, OCP/HRT, pregnancy
Unprovoked VTE: idiopathic or undiagnosed underlying hypercoagulable state or occult malignancy
Most Common DDx
Cellulitis (leg erythema + warmth; fever; WBC elevated; bilateral less common; no Doppler thrombus; responds to antibiotics — can coexist with DVT)
Musculoskeletal injury — Baker's cyst rupture (posterior knee swelling; US shows cyst rupture not DVT; no anticoagulation needed)
Heart failure (bilateral leg edema; elevated BNP; JVD; pulmonary edema on CXR; improves with diuresis)
Pneumonia (PE mimic — dyspnea + pleuritic pain; fever + productive cough; consolidation on CXR; no filling defect on CTA; responds to antibiotics)
Acute coronary syndrome (PE and ACS both cause chest pain + dyspnea; EKG changes; troponin; echo may help differentiate right heart strain vs. LV dysfunction)
Aortic dissection (sudden severe tearing chest/back pain; pulse differential; CT aortography; D-dimer markedly elevated — does not help distinguish from PE)
COPD exacerbation / reactive airway disease (dyspnea + hypoxia; wheeze; responds to bronchodilators; CTA may be needed to exclude PE if pre-test probability intermediate-high)
DATA
Pre-test probability scoring: Wells score for DVT and PE; PERC rule (if all 8 criteria met → PE excluded without further testing)
D-dimer (high sensitivity, low specificity; if low pre-test probability + negative D-dimer → VTE excluded; do not use D-dimer in high pre-test probability — go straight to imaging)
Compression ultrasound (CUS) — first-line for DVT; sensitivity ~95% for proximal DVT; lower for distal
CT pulmonary angiography (CTPA) — gold standard for PE; first-line if intermediate-high probability or PERC positive
V/Q scan — alternative to CTPA if renal insufficiency or contrast allergy; less useful with baseline pulmonary disease
EKG (sinus tachycardia most common; S1Q3T3 pattern, new RBBB, T-wave inversions V1–V4 — right heart strain in massive/submassive PE)
Troponin + BNP/NT-proBNP (right ventricular injury markers — elevated = submassive/massive PE; prognostic)
Echo (RV dilation, RV hypokinesis, D-sign, McConnell's sign — right heart strain in massive PE; valuable for hemodynamically unstable patients)
CBC, BMP, coagulation panel (pre-anticoagulation baseline)
Thrombophilia workup: factor V Leiden, prothrombin gene mutation, protein C/S, antithrombin III, antiphospholipid antibodies (do NOT test while anticoagulated or acutely — affects results)
Age-appropriate malignancy screening if unprovoked VTE (CT chest/abd/pelvis, PSA, mammogram, Pap)
Home Meds
Prior anticoagulation (assess compliance and type — therapeutic vs. prophylactic failure)
OCP/HRT (hold — thrombogenic; switch contraception)
Aspirin / antiplatelets (note — do not rely on aspirin for VTE prevention)
Hormone therapy or selective estrogen receptor modulators (tamoxifen, raloxifene — VTE risk)
Plan
PE risk stratification:
Massive PE (hemodynamic instability — SBP <90 or vasopressors required) → systemic thrombolysis or catheter-directed therapy
Submassive PE (hemodynamically stable + RV dysfunction on echo or troponin/BNP elevated) → anticoagulation ± escalation; ICU/step-down monitoring
Low-risk PE (PESI class I–II, no RV dysfunction, normal troponin/BNP) → consider outpatient anticoagulation
Anticoagulation (first-line for most DVT and non-massive PE):
Apixaban (Eliquis) 10 mg PO BID × 7 days → 5 mg PO BID (preferred DOAC — lowest bleeding risk; AMPLIFY trial)
Rivaroxaban (Xarelto) 15 mg PO BID × 21 days → 20 mg PO daily with evening meal
Dabigatran (Pradaxa) 150 mg PO BID after 5–10 days parenteral anticoagulation
Warfarin: bridge with LMWH × 5–7 days until INR 2–3 × 24h; target INR 2–3; less preferred for initial therapy
Enoxaparin (LMWH): 1 mg/kg SQ q12h or 1.5 mg/kg SQ daily — use in cancer-associated VTE (preferred over DOACs for most malignancy) or renal failure monitoring situations
UFH IV: for massive PE or high bleeding risk requiring rapid reversal; weight-based protocol
Massive PE — thrombolysis:
Alteplase (tPA) 100 mg IV over 2h (systemic) — if SBP <90 refractory to fluids; high bleeding risk (recent surgery, stroke <3 months = absolute contraindication)
Catheter-directed thrombolysis (CDT) or EKOS ultrasound-assisted: submassive or massive PE with contraindications to systemic tPA; IR consult
Surgical embolectomy: if tPA contraindicated or failed; cardiac surgery consult
Duration of anticoagulation:
Provoked (reversible risk factor — surgery/immobility): 3 months
Unprovoked first episode: ≥3 months; individualize extended therapy (low bleeding risk → extended anticoagulation reduces recurrence)
Cancer-associated VTE: indefinite or until cancer resolved; LMWH or DOACs (apixaban/rivaroxaban preferred for most solid tumor cancers)
Recurrent unprovoked VTE: indefinite anticoagulation
Antiphospholipid syndrome: indefinite warfarin (INR 2–3); DOACs inferior in APS
IVC filter: only if absolute contraindication to anticoagulation AND active bleeding; retrieve when anticoagulation resumes (not permanent unless truly lifelong contraindication)
Pulmonary Embolism Response Team (PERT) activation if massive or submassive PE — multidisciplinary
Oxygen supplementation; analgesics for pleuritic pain; IV fluids (cautious — RV preload-dependent)
Hold OCP/HRT; counsel on VTE prevention strategies
Trend CBC, BMP, troponin, BNP; daily if submassive/massive
Hematology consult for thrombophilia workup and extended anticoagulation decisions
PT/OT; compression stockings for DVT
Discharge: DOAC or warfarin with clear duration plan; INR goal and monitoring schedule if warfarin; hold OCP/HRT; malignancy workup if unprovoked; thrombophilia testing 3 months after stopping anticoagulation; hematology follow-up; compression stockings for DVT for symptom management
Red Flags
Massive PE (SBP <90, HR >100, syncope, O2 sat <90%) → ICU; systemic tPA vs. CDT vs. surgical embolectomy urgently
Cardiac arrest from massive PE → thrombolysis during CPR; extended resuscitation (60–90 min) if tPA given
Phlegmasia cerulea dolens (massive DVT with limb ischemia — blue, swollen, painful leg) → systemic tPA or CDT emergently; limb viability threatened
PE in pregnancy → LMWH only (DOACs and warfarin contraindicated); CTPA preferred over V/Q if CXR abnormal; IR/MFM consult
Heparin-induced thrombocytopenia (HIT) with new DVT/PE → stop all heparin; start argatroban or bivalirudin immediately (see HIT section)
Senior IM Resident Pearls
PERC rule: If ALL 8 criteria met (age <50, HR <100, SpO2 ≥95%, no leg swelling, no hemoptysis, no recent surgery/trauma, no prior VTE, no OCP/HRT/estrogen) → PE ruled out without D-dimer in low pre-test probability; do not use if moderate/high pre-test probability
Wells PE score: <2 = low probability; 2–6 = moderate; >6 = high — use with D-dimer and CTPA to guide workup
S1Q3T3 on EKG is classic but only present in ~10–20% of PE — sinus tachycardia + T-wave inversions V1–V4 is more common and more specific for right heart strain
McConnell's sign on echo: RV free wall hypokinesis with preserved RV apex — highly specific for acute PE (but low sensitivity)
DOACs are preferred over warfarin for most VTE — no bridging needed, fixed dosing, predictable pharmacokinetics; exception: APS (DOACs inferior — warfarin preferred), pregnancy (LMWH only), mechanical heart valves
Common mistake: Ordering D-dimer in a patient with high pre-test probability — D-dimer is a rule-out tool only in low-intermediate probability; in high probability patients, go directly to CTPA regardless of D-dimer result
Common mistake: Using systemic tPA for submassive PE routinely — increased bleeding risk; reserve for hemodynamic deterioration; monitor and escalate if RV function worsens
Labs
Admission
CBC with diff
BMP
CMP
PT/INR
PTT
Troponin
BNP or NT-proBNP
Type & Screen (if high-risk PE)
Additional
D-dimer (ONLY if low/intermediate pre-test probability)
ABG/VBG (severe hypoxia)
Lactate (shock/massive PE)
Trending
CBC daily
BMP daily
Troponin q6–8h (submassive/massive PE)
BNP daily (submassive/massive PE)
Imaging
DVT
Venous Duplex Ultrasound
Evaluate:
Proximal DVT
Distal DVT
Clot burden
PE
CT Pulmonary Angiography (CTPA)
First-line diagnostic test
V/Q Scan
If:
Contrast allergy
Severe CKD
Pregnancy consideration
RV Strain Assessment
Echocardiogram
Evaluate:
RV dilation
RV dysfunction
McConnell sign
Pulmonary pressures
Additional
EKG
Evaluate:
Sinus tachycardia
S1Q3T3
RV strain
New RBBB
CXR
Exclude:
Pneumonia
CHF
Pneumothorax
Procedures
Massive PE
Systemic Thrombolysis
Alteplase (tPA) 100 mg IV over 2 hr
Catheter-Directed Thrombolysis (EKOS)
IR consult
Surgical Embolectomy
Cardiac surgery consult
IVC Filter
Only if:
Active VTE
Absolute contraindication to anticoagulation
Medications
Anticoagulation (Preferred)
Apixaban
10 mg PO BID x7 days
Then 5 mg PO BID
Rivaroxaban
15 mg PO BID x21 days
Then 20 mg PO daily
Dabigatran
150 mg PO BID
After 5–10 days of heparin
LMWH
Enoxaparin
1 mg/kg SQ q12h
OR
1.5 mg/kg SQ daily
Preferred:
Cancer-associated VTE
Pregnancy
Some CKD patients
Unfractionated Heparin
Heparin Drip
Weight-based protocol
Preferred:
Massive PE
High bleeding risk
Potential procedures
Need rapid reversal
Massive PE
Alteplase
100 mg IV over 2 hr
If:
SBP <90
Obstructive shock
Oxygen
Supplemental O2
Goal SpO₂ >92%
Pain Control
Acetaminophen
650 mg PO q6h PRN
Oxycodone
2.5–5 mg PO q4–6h PRN
Avoid excessive IV fluids in RV failure.
Hold
OCPs
HRT
Estrogen-containing medications
Tamoxifen if possible
Consults
Hematology
Unprovoked VTE
Recurrent VTE
Thrombophilia evaluation
Pulmonology
Large PE
RV strain
PERT Team
Submassive PE
Massive PE
Interventional Radiology
CDT/EKOS
IVC filter
Cardiac Surgery
Surgical embolectomy candidate
PT/OT
Mobility
Deconditioning
Nursing
Telemetry
Continuous pulse oximetry
Fall precautions
Ambulation as tolerated
Compression stockings (symptom relief)
Notify physician:
Hypotension
New hypoxia
Chest pain
Syncope
Hemoptysis
Follow-Up
CBC daily
BMP daily
Troponin/BNP trend
Monitor oxygen requirements
Monitor bleeding
Review imaging results
Escalation
Massive PE
Signs:
SBP <90
Vasopressor requirement
Syncope
Cardiac arrest
→ ICU
→ tPA
→ PERT
→ IR/Cardiac Surgery
Submassive PE
Signs:
Normal BP
RV dysfunction
Elevated troponin/BNP
→ Stepdown/ICU
→ Echo
→ PERT evaluation
Phlegmasia Cerulea Dolens
Signs:
Massive swollen blue leg
Limb ischemia
→ Vascular Surgery
→ IR
→ Emergent thrombolysis/thrombectomy
HIT Concern
Signs:
Platelets ↓ >50%
New thrombosis
→ Stop all heparin
Argatroban
OR
Bivalirudin
Discharge
Anticoagulation Duration
Provoked VTE
3 months
Unprovoked VTE
≥3 months
Consider indefinite therapy
Cancer-Associated
Indefinite while active cancer
APS
Warfarin lifelong
INR goal 2–3
Follow-Up
Hematology
PCP
Pulmonology if large PE
Counseling
Avoid estrogen-containing medications
Early ambulation
Compression stockings for DVT symptoms