DISEASES: CRITERIA · DIAGNOSTICS · TREATMENT
1. PEPTIC ULCER DISEASE (PUD)
Definition/Criteria: Mucosal break ≥5mm in stomach or duodenum extending through muscularis mucosae; caused by H. pylori (most common), NSAIDs, or acid hypersecretion; duodenal ulcers almost never malignant — gastric ulcers MUST be biopsied
Diagnostics: EGD (gold standard); urea breath test or stool antigen for H. pylori (4 weeks after PPI); BUN:Cr >20 suggests upper GI source; Forrest classification on EGD (Ia = spurting → highest rebleed risk)
Treatment: PPI 40 mg PO BID x4–8 weeks (duodenal), 8–12 weeks (gastric); H. pylori eradication: bismuth quadruple (Omeprazole + Bismuth + Tetracycline + Metronidazole x14 days) or clarithromycin triple if local resistance <15%; hold NSAIDs; EGD follow-up at 8–12 weeks for gastric ulcers
2. GASTRITIS / EROSIVE ESOPHAGITIS
Definition/Criteria: Gastritis = inflammatory mucosal injury of the stomach; erosive esophagitis = mucosal injury of the esophagus; Los Angeles grading A–D for esophagitis (Grade C/D needs BID PPI); H. pylori positive more specific to PUD; erosive esophagitis is endoscopic, GERD is clinical
Diagnostics: EGD; CBC, BMP; H. pylori testing; anti-parietal cell antibody if autoimmune gastritis suspected (+ B12/intrinsic factor Abs); Grade A/B → daily PPI; Grade C/D → BID PPI
Treatment: Gastritis: PPI BID x4–8 weeks; H. pylori eradication if positive; avoid NSAIDs/alcohol; Erosive esophagitis: Pantoprazole 40 mg BID x8 weeks (LA grade C/D), step down to daily PPI; Eosinophilic esophagitis (EoE): budesonide oral suspension 2 mg BID x12 weeks OR swallowed fluticasone; 6-food elimination diet
3. GERD / ESOPHAGEAL MOTILITY DISORDERS
Definition/Criteria: GERD = chronic acid reflux causing symptoms or mucosal injury; diagnosed clinically or pH monitoring; EGD may be normal; Achalasia = aperistalsis + incomplete LES relaxation (barium "bird's beak"); distinguish from cancer by age, manometry, and EGD
Diagnostics: EGD first (exclude Barrett's, malignancy); manometry for achalasia (gold standard — aperistalsis + incomplete LES relaxation); pH monitoring for GERD without esophagitis; barium swallow for stricture/achalasia; biopsy ≥15 eos/hpf = EoE
Treatment: GERD: lifestyle modification + PPI daily; Achalasia: pneumatic dilation or POEM (preferred for younger); nifedipine 10–20 mg SL before meals as bridge; EoE: budesonide oral suspension + 6-food elimination diet, PPI trial first; Stricture: endoscopic dilation q4–6 weeks PRN; Malignancy: CT staging + multidisciplinary oncology referral
4. MALLORY-WEISS TEAR
Definition/Criteria: Longitudinal mucosal laceration at the GEJ from forceful vomiting or retching; NO precipitating retching = think Boerhaave; mucosal only (not full thickness); EGD shows mucosal tear at GEJ on retroflexion; usually self-limited (>90% stop spontaneously)
Diagnostics: EGD (retroflexion at GEJ — key view); CBC, BMP, coagulation; always scope to confirm source; distinguish from varices, PUD, Boerhaave; no free air on imaging (distinguishes from perforation)
Treatment: >90% spontaneous resolution; IV access + fluid resuscitation; NPO until EGD; PPI IV; endoscopic hemostasis (epinephrine injection/clips) if active bleeding; treat with antiemetics (ondansetron 4 mg IV q6h, promethazine 12.5 mg IV q6h PRN); alcohol cessation; discharge with PPI x4–8 weeks; follow-up 2–4 weeks
5. BOERHAAVE SYNDROME (ESOPHAGEAL PERFORATION)
Definition/Criteria: Full-thickness esophageal rupture from sudden forceful vomiting; sudden tearing chest/back pain + subcutaneous emphysema + mediastinal air = surgical emergency; distinguish from Mallory-Weiss (mucosal only, no free air, less severe)
Diagnostics: CXR (mediastinal widening, subcutaneous emphysema, pleural effusion — usually left); CT chest with oral contrast (confirms perforation, mediastinal air); avoid EGD initially; left pleural effusion with low pH = amylase-rich fluid (diagnostic)
Treatment: Surgical emergency — immediate surgical consult; NPO, broad-spectrum antibiotics (pip-tazo), IV PPI; early surgical repair (<24h = best outcomes); late presentation: esophageal stenting + drainage; never delay for further workup if clinically suspected
6. VARICEAL BLEEDING (ESOPHAGEAL/GASTRIC)
Definition/Criteria: Hemorrhage from portal hypertension-driven esophageal or gastric varices; stigmata of liver disease + large-volume hematemesis; HR >100 + SBP <90 = ICU; gastric varices bleed more, are harder to treat than esophageal; cirrhosis history or signs (spider angiomata, splenomegaly, ascites)
Diagnostics: EGD within 12h (EVL = endoscopic variceal ligation is first-line); CBC q6h, BMP, LFTs, coags, type & screen; trend ammonia + fever; restrictive transfusion (Hgb 7–8, platelets <50k = airway protection before EGD); INR unreliable in cirrhosis (use TEG)
Treatment: Octreotide 50 mcg IV bolus → 50 mcg/hr x3–5 days; ceftriaxone 1g IV daily x7 days (SBP prophylaxis — reduces mortality); EVL at EGD; early TIPS (<72h) if Child B active bleed or Child C <14 pts; balloon tamponade only as bridge; restrictive transfusion (Hgb 7–8); non-selective BB (propranolol or carvedilol) after acute bleed; avoid beta-blockers acutely (blunts compensatory tachycardia)
7. NON-VARICEAL UPPER GI BLEEDING (NVUGIB)
Definition/Criteria: Upper GI bleed from non-variceal source (PUD most common, also Mallory-Weiss, GAVE, Dieulafoy); Forrest classification drives management: Ia (spurting) → highest rebleed risk → IV PPI infusion; BUN:Cr >20 suggests upper GI source; melena = upper GI until proven otherwise
Diagnostics: EGD within 24h (hemoclip or epinephrine if active bleed/visible vessel); CBC, BMP, coags, type & screen; BUN:Cr ratio; H. pylori testing; NG lavage not routinely required
Treatment: IV PPI bolus 80 mg → 8 mg/hr infusion x72h (high-risk lesion); EGD hemostasis (epinephrine + clip or thermal); IV access x2, resuscitate; NPO; antiemetics; failed endoscopic hemostasis x2 → IR embolization → surgery; discharge: PPI daily x4–8 weeks; hold NSAIDs; H. pylori eradicate if positive
8. GASTRIC ANTRAL VASCULAR ECTASIA (GAVE) / PORTAL HYPERTENSIVE GASTROPATHY (PHG)
Definition/Criteria: GAVE = "watermelon stomach" — discrete vascular ectasias in antrum (not portal HTN driven); PHG = diffuse mucosal congestion in cirrhosis ("snake skin"/"mosaic" pattern) from portal HTN; both cause chronic occult/overt bleeding and iron deficiency anemia; treated differently — GAVE uses APC, NOT banding
Diagnostics: EGD (GAVE: discrete red spots in antrum in watermelon pattern; PHG: proximal stomach diffuse congestion); CBC (iron deficiency anemia); iron studies; liver disease workup (if PHG suspected); multiple EGDs often needed for GAVE
Treatment: GAVE: argon plasma coagulation (APC) at EGD; iron supplementation; transfusion if anemic; thalidomide or hormonal therapy for refractory; PHG: non-selective BB (propranolol/carvedilol) to reduce portal pressure; TIPS for refractory PHG; iron supplementation
9. DIEULAFOY LESION
Definition/Criteria: Large submucosal artery eroding through mucosa without ulceration; recurrent large-volume GI bleeds with no ulcer found; most common in proximal stomach (within 6 cm of GEJ); EGD may miss it — requires multiple endoscopies; NO prior retching, NO liver disease, NO ulcer = think Dieulafoy
Diagnostics: EGD (may need multiple scopes — can be very small, easily missed); tagged RBC scan or CT angiography if EGD negative; push enteroscopy if suspected small bowel; lesion appears as pulsatile artery or visible vessel without surrounding ulcer
Treatment: Endoscopic hemostasis (hemoclip preferred — best long-term results); epinephrine injection + clip or thermal; IR embolization if endoscopy fails; surgery (wedge resection) for refractory; PPI after; consider recurrent bleeds = always biopsy at EGD, always scope for source
10. AORTOENTERIC FISTULA
Definition/Criteria: Communication between aorta (or aortic graft) and GI tract (usually duodenum); prior aortic surgery/graft = must rule out; herald bleed (small bleed followed by exsanguinating hemorrhage) is classic; massive hemorrhage after any prior aortic graft surgery = aortoenteric fistula until proven otherwise
Diagnostics: CT angiography (imaging of choice — shows graft, perigraft air, fistula tract); EGD if stable (shows blood in distal duodenum, may see graft); DO NOT delay CT for EGD if prior aortic graft + massive bleed
Treatment: Surgical emergency — immediate vascular surgery consult; IV access, resuscitate, crossmatch; broad-spectrum antibiotics; endovascular stenting as bridge to surgery; high mortality even with surgery; never manage conservatively
11. FOOD BOLUS IMPACTION / ESOPHAGEAL FOREIGN BODY
Definition/Criteria: Difficulty swallowing or complete obstruction by food bolus requiring urgent endoscopic removal; EoE is #1 cause in young adults (always biopsy even if esophagus looks normal); esophageal malignancy in elderly (weight loss, progressive dysphagia); impacted >6h = risk of pressure necrosis
Diagnostics: CXR (exclude free air or mediastinal widening — if present = perforation → surgery first); CT if perforation suspected; EGD = diagnostic and therapeutic; glucagon 1 mg IV can relax LES and allow passage; always biopsy after removal
Treatment: Urgent EGD within 6h (drooling/inability to manage secretions) or 24h (partial); push or pull technique; NPO; post-removal inspection + biopsy for underlying lesion; treat EoE if found; PPI ongoing for stricture; Zenker's diverticulum: alert endoscopist before EGD (perforation risk with blind intubation); Surgery if endoscopy fails or perforation
12. ANEMIA OF GI ETIOLOGY (IRON DEFICIENCY / HEMORRHAGIC)
Definition/Criteria: Anemia from acute hemorrhage (normocytic, hemodilution takes 24–48h) or chronic GI blood loss (microcytic, low ferritin, low MCV); iron deficiency anemia in adults = GI malignancy until proven otherwise; distinguish from anemia of chronic disease (normal/high ferritin, low TIBC) and hemolytic (elevated LDH, indirect bili, low haptoglobin)
Diagnostics: CBC, reticulocyte count, iron studies (serum iron, ferritin, TIBC, transferrin sat); peripheral smear; BMP; hemolysis panel (LDH, haptoglobin, indirect bili, Coombs) if hemolytic suspected; EGD ± colonoscopy for source identification; serial CBC q6h if acute
Treatment: Transfusion thresholds: Hgb <7 (stable, non-cardiac); Hgb <8 (ACS, hemodynamic instability, elderly with cardiovascular disease); 1 unit pRBCs raises Hgb ~1 g/dL; IV iron (ferric carboxymaltose 500–1000 mg) if deficient and PO not tolerated; ferrous sulfate 325 mg PO TID with vitamin C; correct coagulopathy (FFP if INR >1.5 + bleeding); identify and treat source
PART 2 — DDX QUICK REVIEW
HEMATEMESIS / UPPER GI BLEED
PUD (Bleeding)
Epigastric pain, NSAID use, H. pylori history, melena, BUN:Cr >20
Dx: EGD (Forrest classification), H. pylori testing
Tx: IV PPI (80 mg bolus → 8 mg/hr infusion), endoscopic hemostasis, eradicate H. pylori
Variceal Bleed
Cirrhosis, portal HTN, splenomegaly, large-volume hematemesis
Dx: EGD within 12 hr
Tx: Octreotide + Ceftriaxone + EVL ± TIPS
Mallory-Weiss Tear
Hematemesis after forceful retching/vomiting
No mediastinal air
Dx: EGD (retroflexion)
Tx: Antiemetics, PPI, endoscopic therapy if active bleeding
Boerhaave Syndrome
Vomiting → sudden chest/back pain + subcutaneous emphysema
Mediastinal air present
Dx: CT chest with oral contrast
Tx: Surgical emergency, broad-spectrum antibiotics, repair <24 hr
Dieulafoy Lesion
Massive recurrent bleeding
No ulcer, no cirrhosis
Often multiple negative EGDs
Dx: Repeat EGD ± CTA
Tx: Hemoclip, IR embolization if needed
Aortoenteric Fistula
Prior aortic graft + herald bleed
Dx: CT angiography FIRST
Tx: Emergent vascular surgery
GAVE
Chronic iron deficiency anemia
"Watermelon stomach"
Dx: EGD
Tx: APC (NOT banding)
Upper GI Malignancy
Weight loss, dysphagia, age >50
Irregular ulcer margins
Dx: EGD + biopsy, CT staging
Tx: Oncology referral
DYSPHAGIA
Eosinophilic Esophagitis (EoE)
Young male, atopy, food impaction
Dx: EGD + biopsy (≥15 eos/hpf)
Tx: Budesonide + 6-food elimination diet + PPI trial
Esophageal Stricture
Progressive solids > liquids
Long-standing GERD
Dx: EGD or barium swallow
Tx: Endoscopic dilation + PPI
Achalasia
Progressive solids AND liquids
Regurgitation of undigested food
Bird's-beak appearance
Dx: Manometry (gold standard)
Tx: POEM or pneumatic dilation
Esophageal Cancer
Progressive dysphagia
Weight loss
Smoker/alcohol history
Dx: EGD + biopsy
Tx: Oncology, surgery ± chemo/XRT
Zenker Diverticulum
Halitosis
Regurgitation of undigested food
Coughing/choking
Dx: Barium swallow
Tx: Surgical/endoscopic repair
Oropharyngeal Dysphagia
Choking immediately upon swallowing
Nasal regurgitation
Neurologic disease
Dx: Video swallow study
Tx: SLP therapy + treat underlying cause
Schatzki Ring
Intermittent solid-food dysphagia
Normal between episodes
Dx: EGD or barium swallow
Tx: Endoscopic dilation
EPIGASTRIC PAIN
PUD
Duodenal ulcer: relieved by food
Gastric ulcer: worsened by food
Melena, NSAIDs, H. pylori
Dx: EGD + H. pylori testing
Tx: PPI + eradicate H. pylori
GERD / Esophagitis
Burning substernal pain
Worse lying down
Improved with antacids
Dx: Clinical ± EGD
Tx: PPI + lifestyle modification
Acute Pancreatitis
Epigastric pain radiating to back
Lipase >3× ULN
Dx: Lipase
Tx: IVF, analgesia, early enteral feeding
Biliary Colic
Post-fatty meal RUQ/epigastric pain
Dx: RUQ ultrasound
Tx: Analgesics, elective cholecystectomy
Acute Cholecystitis
Fever + RUQ pain + Murphy sign
Dx: RUQ ultrasound
Tx: Antibiotics + cholecystectomy
Functional Dyspepsia
Chronic symptoms
Normal EGD
Dx: Diagnosis of exclusion
Tx: PPI ± low-dose TCA
Gastric Cancer
Weight loss
Early satiety
Age >50
Dx: EGD + biopsy
Tx: Oncology referral
Perforated Viscus
Sudden severe pain
Rigid abdomen
Free air
Dx: CXR/CT
Tx: Emergent surgery
ANEMIA (GI CONTEXT)
Iron Deficiency Anemia
Microcytic anemia
Low ferritin
Chronic occult GI blood loss
Dx: Iron studies + GI workup
Tx: Iron replacement + identify source
Anemia of Chronic Disease
Normocytic anemia
High ferritin, low TIBC
Dx: Iron studies
Tx: Treat underlying disease
B12/Folate Deficiency
Macrocytic anemia
Hypersegmented neutrophils
Neurologic symptoms (B12)
Dx: B12/Folate levels
Tx: Vitamin replacement
Hemolytic Anemia
↑ LDH, ↑ indirect bilirubin, ↓ haptoglobin
Dx: Hemolysis labs
Tx: Treat underlying cause
Acute Hemorrhagic Anemia
Acute GI bleed
Tachycardia, hypotension
Hgb may initially be normal
Dx: Serial CBCs
Tx: Resuscitation + transfusion
Aplastic Anemia
Pancytopenia
Hypocellular marrow
Dx: Bone marrow biopsy
Tx: Hematology referral
ACUTE GI BLEED — RAPID TRIAGE
Hematemesis + Cirrhosis + Unstable → Variceal bleed → ICU + EGD ≤12 hr
Melena + NSAIDs + Epigastric pain → PUD → PPI drip + EGD ≤24 hr
Retching → Hematemesis → Mallory-Weiss tear
Vomiting → Chest pain + Crepitus → Boerhaave → CT chest → Surgery NOW
Prior Aortic Graft + GI Bleed → Aortoenteric fistula → CTA → Vascular surgery NOW
Recurrent Massive Bleed + Negative EGD → Dieulafoy lesion
Iron Deficiency + Watermelon Stomach → GAVE → APC
Young Male + Food Impaction + Atopy → EoE → EGD + biopsy + budesonide
This format reads much faster during rounds and call nights than the original table.